Abstract
Cushing’s disease (CD) is the most common cause of endogenous cortisol excess. We discuss the case of a 60-year-old woman with recurrent venous thromboembolism, refractory hypokalemia, and lumbar vertebrae compression fractures with a rapidly progressive disease course. Ectopic hypercortisolism was suspected given the patient’s age and rapid onset of disease. Investigations revealed cortisol excess from a pituitary microadenoma. This case demonstrates that CD can present with severe findings and highlights the increased risk of venous thromboembolism in hypercortisolism, especially in CD.
Original language | English (US) |
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Pages (from-to) | 715-717 |
Number of pages | 3 |
Journal | Baylor University Medical Center Proceedings |
Volume | 34 |
Issue number | 6 |
DOIs | |
State | Published - 2021 |
Externally published | Yes |
Keywords
- Cortisol
- Cushing’s disease
- fracture
- hypercoagulability
- thromboembolism
ASJC Scopus subject areas
- General Medicine