Original language | English (US) |
---|---|
Pages (from-to) | 1-3 |
Number of pages | 3 |
Journal | Journal of Cell Biology |
Volume | 217 |
Issue number | 1 |
DOIs | |
State | Published - Jan 1 2018 |
Externally published | Yes |
ASJC Scopus subject areas
- Cell Biology
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New editorial board members. / Balla, Tamas; Baccarini, Manuela; Barr, Maureen et al.
In: Journal of Cell Biology, Vol. 217, No. 1, 01.01.2018, p. 1-3.Research output: Contribution to journal › Editorial › peer-review
}
TY - JOUR
T1 - New editorial board members
AU - Balla, Tamas
AU - Baccarini, Manuela
AU - Barr, Maureen
AU - Castellani, Valérie
AU - Cimini, Daniela
AU - Ephrussi, Anne
AU - Etienne-Manneville, Sandrine
AU - Haigis, Marcia
AU - Kapoor, Tarun
AU - Leschziner, Andres
AU - Manning, Brendan D.
AU - Meyer, Tobias
AU - Miller, Liz
AU - Nachury, Maxence
AU - Prinz, Will
AU - Smogorzewska, Agata
AU - Torres-Padilla, Maria Elena
N1 - Funding Information: Dr. Maxence (Max) Nachury studied biology and biochemistry at the École Normale Supérieure (Paris) before joining the laboratory of Karsten Weis (first at University of California, San Francisco, and then at University of California, Berkeley) for his PhD. There, he studied the role of the small GTPase Ran in enforcing the directionality of nucleocytoplasmic transport and in chromatin-driven self-organization of the mitotic spindle (in collaboration with Rebecca Heald). For his postdoc, Max moved to Stanford University and then Genentech to work with Peter Jackson on the cell cycle before switching his interests toward the primary cilium. Since starting his research group at Stanford University in 2008, his laboratory has worked to disentangle the molecular basis of Bardet-Biedl Syndrome (BBS), a prototypical ciliopathy characterized by obesity, retinal degeneration, polydactyly, and cystic kidneys. Through biochemical reconstitution, single molecule imaging of signaling receptor dynamics, and in situ proteomics, his laboratory has established that the BBSome, a stable complex of eight BBS proteins, moves membrane proteins out of cilia by ferrying them across the transition zone, a diffusion barrier at the base of cilia. Other interests of his laboratory include signal-dependent ectocytosis, a novel mechanism that packages activated signaling receptors into extracellular vesicles at the tip of cilia, tubulin acetylation, and centriole regulation. He moved his laboratory to the University of California, San Francisco, in 2017. Dr. Nachury has been the recipient of a Sloan Fellowship, a Searle Scholar Award, a Klingenstein Fellowship, the ASCB Early Career Life Scientist Award, and a Keith Porter Fellowship. Photo courtesy matthew scott.
PY - 2018/1/1
Y1 - 2018/1/1
UR - http://www.scopus.com/inward/record.url?scp=85039857051&partnerID=8YFLogxK
UR - http://www.scopus.com/inward/citedby.url?scp=85039857051&partnerID=8YFLogxK
U2 - 10.1083/jcb.201712071
DO - 10.1083/jcb.201712071
M3 - Editorial
AN - SCOPUS:85039857051
SN - 0021-9525
VL - 217
SP - 1
EP - 3
JO - Journal of Cell Biology
JF - Journal of Cell Biology
IS - 1
ER -