Macrophages regulate gastrointestinal motility through complement component 1q

Mihir Pendse, Haley De Selle, Nguyen Vo, Gabriella Quinn, Chaitanya Dende, Yun Li, Cristine N. Salinas, Tarun Srinivasan, Daniel C. Propheter, Alexander A. Crofts, Eugene Koo, Brian Hassell, Kelly A. Ruhn, Prithvi Raj, Yuuki Obata, Lora V. Hooper

Research output: Contribution to journalArticlepeer-review

7 Scopus citations

Abstract

Peristaltic movement of the intestine propels food down the length of the gastrointestinal tract to promote nutrient absorption. Interactions between intestinal macrophages and the enteric nervous system regulate gastrointestinal motility, yet we have an incomplete understanding of the molecular mediator’s of this crosstalk. Here we identify complement component Iq (Clq) as a macrophage product that regulates gut motility. Macrophages were the predominant source of Clq in the mouse intestine and most extraintestinal tissues. Although Clq mediates complement-mediated killing of bacteria in the bloodstream, we found that Clq was not essential for immune defense of the intestine. Instead. Clq- expressing macrophages were located in the intestinal submucosal and myenteric plexuses where they closely associated with enteric neurons and expressed surface markers characteristic of nerve-adjacent macrophages in other tissues. Mice with a macrophage-specific deletion of Clqa showed changes in enteric neuronal gene expression, increased neurogenic activity of peristalsis, and accelerated intestinal transit.

Original languageEnglish (US)
Article numbere78558
JournaleLife
Volume12
DOIs
StatePublished - Apr 2023

ASJC Scopus subject areas

  • General Neuroscience
  • General Biochemistry, Genetics and Molecular Biology
  • General Immunology and Microbiology

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