Abstract
A by-product of cancer (and of other chronic diseases or severe trauma), cachexia appears to be mediated by various cytokines. The first to be identified-tumor necrosis factor-may be a principal mediator. With molecular cloning techniques, future treatment strategies could include blockade of TNF activity using highly specific reagents.
| Original language | English (US) |
|---|---|
| Pages (from-to) | 45-52 |
| Number of pages | 8 |
| Journal | Hospital Practice |
| Volume | 28 |
| Issue number | 4 |
| DOIs | |
| State | Published - 1993 |
ASJC Scopus subject areas
- General Medicine