Correction: Epigenetic loss of AOX1 expression via EZH2 leads to metabolic deregulations and promotes bladder cancer progression (Oncogene, (2020), 39, 40, (6265-6285), 10.1038/s41388-019-0902-7)

Venkatrao Vantaku, Vasanta Putluri, David A. Bader, Suman Maity, Jing Ma, James M. Arnold, Kimal Rajapakshe, Sri Ramya Donepudi, Friedrich Carl von Rundstedt, Vaishnavi Devarakonda, Julien Dubrulle, Balasubramanyam Karanam, Sean E. McGuire, Fabio Stossi, Abhinav K. Jain, Cristian Coarfa, Qi Cao, Andrew G. Sikora, Hugo Villanueva, Shyam M. KavuriYair Lotan, Arun Sreekumar, Nagireddy Putluri

Research output: Contribution to journalComment/debatepeer-review

2 Scopus citations

Abstract

After publication of this Article, the Authors noticed errors in some of the Figures. In Figures 2e, 2f–g, 4a, 4j, 5a and 6b, unmatched β-actin was inadvertently used as loading control for the immunoblots. These have been corrected using repeat data from a similar set of samples and the revised Figures containing matched β-actin and their respective quantification data are included below. In Figure 7a, the same image was inadvertently used to represent tumors 3 and 5 in the control group. This error has been corrected using original images of tumors 3 and 5 in the control group. Additional corrections have been made in the Article and Figure legends to enhance the clarity of the description. NAD was replaced by NADP. NAD/NADP was replaced by NADP/NADPH. The description of the antibody source and dilution for the antigens PFKFB4 (Abcam, 1:1000), G6PD, and HK1 (Cell Signaling, 1:1,000) have been included in the Methods section for Western Blot. The legend for Figure 4e and 4j has been updated. The HTML and PDF versions of this Article have been corrected. The scientific conclusions of this paper have not been affected.

Original languageEnglish (US)
Pages (from-to)6387-6392
Number of pages6
JournalOncogene
Volume39
Issue number40
DOIs
StatePublished - Oct 1 2020

ASJC Scopus subject areas

  • Molecular Biology
  • Genetics
  • Cancer Research

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