Abstract
Coronary artery disease (CAD) that results from lesions of the vascular wall is a major cause of myocardial infarction (MI) and stroke. A human pedigree with a predisposition to CAD and MI has been shown to harbor a mutation in the MEF2A transcription factor. These findings reveal a new function for this regulator of cardiovascular development and raise intriguing questions about the underlying mechanisms of CAD.
| Original language | English (US) |
|---|---|
| Pages (from-to) | pe33 |
| Journal | Science of aging knowledge environment : SAGE KE |
| Volume | 2003 |
| Issue number | 48 |
| State | Published - Dec 3 2003 |
ASJC Scopus subject areas
- Medicine(all)