Adipocyte metabolism and obesity

Alan D. Attie; Philipp E. Scherer

doi:10.1194/jlr.R800057-JLR200

Adipocyte metabolism and obesity

Alan D. Attie, Philipp E. Scherer

Research output: Contribution to journal › Review article › peer-review

165 Scopus citations

Abstract

Adipose tissue metabolism exerts a profound impact on whole-body metabolism. We review how fuel partitioning between adipocytes and other tissues affects insulin signaling pathways. We discuss the role of adipose tissue inflammation in adipocyte metabolism and whole-body insulin sensitivity. Finally, we mention the role of adipokines in autocrine and paracrine signaling.

Original language	English (US)
Pages (from-to)	S395-S399
Journal	Journal of lipid research
Volume	50
Issue number	SUPPL.
DOIs	https://doi.org/10.1194/jlr.R800057-JLR200
State	Published - Apr 2009

Keywords

Adiponectin
Inflammation
Leptin

ASJC Scopus subject areas

Biochemistry
Endocrinology
Cell Biology

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10.1194/jlr.R800057-JLR200

Cite this

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title = "Adipocyte metabolism and obesity",

abstract = "Adipose tissue metabolism exerts a profound impact on whole-body metabolism. We review how fuel partitioning between adipocytes and other tissues affects insulin signaling pathways. We discuss the role of adipose tissue inflammation in adipocyte metabolism and whole-body insulin sensitivity. Finally, we mention the role of adipokines in autocrine and paracrine signaling.",

keywords = "Adiponectin, Inflammation, Leptin",

author = "Attie, {Alan D.} and Scherer, {Philipp E.}",

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AB - Adipose tissue metabolism exerts a profound impact on whole-body metabolism. We review how fuel partitioning between adipocytes and other tissues affects insulin signaling pathways. We discuss the role of adipose tissue inflammation in adipocyte metabolism and whole-body insulin sensitivity. Finally, we mention the role of adipokines in autocrine and paracrine signaling.

KW - Adiponectin

KW - Inflammation

KW - Leptin

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Adipocyte metabolism and obesity

Abstract

Keywords

ASJC Scopus subject areas

Access to Document

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