A NIK–SIX signalling axis controls inflammation by targeted silencing of non-canonical NF-κB

Medicine and Dentistry

• Phosphotransferase 100%
• Transcription Factors 100%
• Disease 50%
• Cell Death 50%
• Ubiquitin 50%
• B Cell 50%
• Non Small Cell Lung Cancer 50%
• Protein P53 50%
• Gene Expression 50%
• Chemotherapy 50%
• Macrophage 50%
• Negative Feedback 50%
• Inflammatory Cell 50%
• Osteoclastogenesis 50%
• Transgene 50%
• Septic Shock 50%
• Gene Promoter 50%
• Cell Maturation 50%
• Proteasome 50%
• Homeobox 50%
• Transactivation 50%
• Immune Disorder 50%
• Lymphoid Organogenesis 50%

Keyphrases

• Inflammation 100%
• NF-B 100%
• Non-canonical 100%
• Signaling Axis 100%
• SIX1 57%
• Six2 42%
• Transcription Factor 28%
• Signaling Cascades 28%
• Non-small Cell Lung Cancer (NSCLC) 14%
• Macrophages 14%
• Ubiquitin-proteasome System 14%
• Human Disease 14%
• Pathological Conditions 14%
• Inflammatory Gene Expression 14%
• Transcriptional Response 14%
• Negative Feedback Loop 14%
• Transactivation 14%
• Physiological Condition 14%
• Host Immunity 14%
• Inflammatory Genes 14%
• Smac Mimetics 14%
• Osteoclast Differentiation 14%
• Endotoxic Shock 14%
• Gene Promoter 14%
• Integral Component 14%
• Rate-limiting Step 14%
• Homeobox 14%
• Gene Expression Programming 14%
• Inflammatory Cell Death 14%
• MAPK14 14%
• B Cell Maturation 14%
• Immunological Disorders 14%
• Small Molecule Activation 14%
• Lymphoid Organogenesis 14%
• Sine Oculis 14%

Biochemistry, Genetics and Molecular Biology

• SIX2 75%